The CuAl5 single crystal with the initial orientation (I 10)[00I] was cold rolled and subsequently compressed in a channel die in such a way that the new direction of plastic flow was parallel to the transverse direction of initial rolling and the compressed plane was parallel to the former rolling plane. The dislocation slip was the only deformation mechanism during rolling. The dislocation substructure after rolling was uniform. During the compression in a channel die, additionally, the deformation twinning as well as the shear band formation took place. The recrystallization commenced and proceded within the shear bands. After that. the recrystallization proceeded by the nucleation and growth of new grains within the areas with high density of deformation twins. The recrystallized regions of the former shear bands were filled by fine grains while the regions amongst them exhibited much bigger grains. The recrystallization texture was formed according to the theory of the oriented growth.

The formation of texture and microstructure in polycrystalline Cu and Cu-5 wt. % Al alloy during two modes of homogenous rolling have been compared. Two samples of each metal were rolled. One sample was rolled unidirectionally, while the other by reverse rolling. Some differences in the textures depending on the rolling mode have been observed in the range of deformation of the Cu-5 wt. % Al alloy where intensive twinning took place. It was also found that the texture of reverse rolling was more homogeneous across the thickness of the rolled alloy sheet than the texture of unidirectionally rolled material. No clear effect of the rolling mode on microstructure has been observed.

The brain is subject to damage, due to ageing, physiological processes and/or disease. Some of the damage is acute in nature, such as strokes; some is more subtle, like white matter lesions. White matter lesions or hyperintensities (WMH) can be one of the first signs of micro brain damage. We implemented the Acoustocerebrography (ACG) as an easy to use method designed to capture differing states of human brain tissue and the respective changes.

Aim: The purpose of the study is to compare the efficacy of ACG and Magnetic Resonance Imaging (MRI) to detect WMH in patients with clinically silent atrial fibrillation (AF).

Methods and results: The study included 97 patients (age 66.26 ± 6.54 years) with AF. CHA2DS2-VASc score (2.5 ±1.3) and HAS BLED (1.65 ± 0.9). According to MRI data, the patients were assigned into four groups depending on the number of lesions: L0 – 0 to 4 lesions, L5 – 5 to 9 lesions, L10 – 10 to 29 lesions, and L30 – 30 or more lesions. Authors found that the ACG method clearly differentiates the groups L0 (with 0–4 lesions) and L30 (with more than 30 lesions) of WMH patients. Fisher’s Exact Test shows that this correlation is highly significant (p < 0:001).

Conclusion: ACG is a new, easy and cost-effective method for detecting WMH in patients with atrial fibrillation